A new study conducted by researchers from the University of Bristol and the Federal University of São Paulo has shown that regular aerobic exercise can significantly reduce key markers of Alzheimer’s disease, including amyloid plaques and tau formations. The experiment on rodents revealed that physical activity helped reduce these pathological changes and also decreased iron accumulation in myelin-producing cells. Animals that engaged in aerobic exercise showed a 63% reduction in tau formations, a 76% reduction in amyloid plaques, and a 58% reduction in iron buildup.
In addition, physical activity led to improvements in brain cell health. The number of protective oligodendrocytes increased, inflammation was reduced by 55-68%, and neuronal activity rose, which improved communication between cells and restored normal brain function. These results highlight the powerful neuroprotective effect of aerobic exercise and emphasize its potential role in preventing and slowing the progression of Alzheimer’s disease.
Dr. Augusto Coppi, senior lecturer at the University of Bristol, noted that aerobic exercise could become an important part of Alzheimer’s treatment, especially given the lack of effective drugs to slow cognitive decline. The research suggests that such exercises may play a key role in preventing neurodegenerative diseases, and researchers are calling for the development of physical activity programs for older adults.
The next step will be to conduct clinical trials on humans to confirm the effectiveness of aerobic exercise in slowing disease progression. Researchers also plan to develop medications aimed at improving metabolism, which could complement preventive measures.
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